As if chronically on Thiazide-like Diuretics

Na-Cl

Gitelman syndrome is a salt-losing tubulopathy caused by mutation of genes encoding sodium chloride (NCCT) and magnesium transporters in the thiazide-sensitive segments of the distal nephron. It is characterized by renal potassium wasting, hypokalemia, metabolic alkalosis, hypocalciuria, hypomagnesemia, and hyperreninemic hyperaldosteronism.

Gitelman syndrome is also referred to as familial hypokalemia-hypomagnesemia. This activity highlights the role of the interprofessional team in managing patients with Gitelman syndrome to provide the best patient outcomes.